Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Conversely, our ability to rescue SERT operate in KI midbrain synaptoneurosomes because of the inhibition of FAK implies elevated FAK signaling downstream from the Pro32Pro33 mutant, as confirmed by improved pFAK localization in 5-HT synapses. https://chrisi777ojd2.blog2freedom.com/profile